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Although clinicians commonly hypothesize an association between the typical American diet and LUTS, 8 empirical data are limited, with most of the existing studies reaching inconclusive or contradictory findings. Associations between longitudinal changes in serum estrogen, testosterone, and bioavailable testosterone and changes in benign urologic outcomes. A role for estrogen receptor beta in the regulation of growth of the ventral prostate. Intakes of energy and macronutrients and the risk of benign prostatic hyperplasia. Each HEI diet variable was analyzed in our models individually because of variable collinearity. Although the causal pathway remains uncertain, it has been hypothesized that a healthier lifestyle may lower the incidence of LUTS through the effect on sympathetic tone, 4 androgen levels, 5 , 6 and inflammation. Autonomic nervous system overactivity in men with lower urinary tract symptoms secondary to benign prostatic hyperplasia. Bivariate analysis of HEI categories and symptoms is shown in Table 2. Comparison of weighted demographic information between those with and without symptoms is shown in Table 1.

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